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This mechanistic proposal was initially suggested because the ACMs appear to possess high iron-binding affinity and because of their involvement in a wide range of interactions with iron-containing proteins and iron metabolic pathways, as well as antioxidant activity and related effects [18]

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Additionally, apoptotic T cells secrete TGF-β which acts as a growth factor for FoxP3 − LAP + Tregs [29, 92, 93] and CD4 + CD25 + Tregs [94–97]